By Dale Brown
In army circles it's referred to as Air conflict strength -- an air strive against unit of the long run, less than the command of aerial struggle professional significant normal Patrick McLanahan, able to launching stealthy, precision-guided firepower to at any place at the globe inside of hours. And now McLanahan and his warriors have their first target.
Driven from Afghanistan, the parasitic forces of the deposed Taliban regime have determined to feed on a brand new host -- an remoted, oil-rich primary Asian neighbor that's ripe for the conquering. The conflict for regulate of the world's biggest oil deposits has began, with unsteady alliances forming and opposing forces set to collide. And now a handful of yankee commandos part an international away, aided by way of McLanahan's unproven robot warplanes, should triumph opposed to overwhelming numbers in a battle that no-one -- together with "friendly" forces at domestic -- desires them to win.
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On the other hand, the cytotoxic activity to K562 was slightly reduced (10--20%), as compared with control cultures (Fig. 4). These results show that endogenously produced IFN--y is essential to the proliferation and cytotoxicity of MLC but not to the lytic activity of natural killer cells, which is responsible for K562-cell killing. By using affinity-purified human antibodies to IFN--y we definitively demonstrated their unique ability to neutralize certain immunomodulating activities of IFN--y but not its antiviral and antiproliferative activity.
The characterization of human anti-cytokine autoantibodies revealed that they are polyclonal in nature and belong almost exclusively to the IgG class (14, 15). Those directed to IL-IO' are for almost 50% of the IgG4 subclass whereas anti-IFN-, and anti-IL6 autoantibodies were characterized as IgG I . Importantly, the serum levels of ACAs can vary significantly during infectious diseases with high levels at the height of the disease and a significant reduction after disease resolution (16). This suggests that when a specific cytokine exceeds a critical treshold level, ACAs may be compensatorily increased.
ALLOTRANSPLANT REJECTION Rejection of tumor, skin and heart allografts delayed by mABs anti-IFN--y. KAPOSI'S SARCOMA (KS) IFN--y induces normal endothelial cells to acquire the phenotype of KS cells. KS lesions blocked by mABs anti-IFN-T flow cytometric assay [17-18]. It remains yet unclear why the neutralizing effects of the immunoglobulins disappeared 13 days after infection, but this may be related either to preferential consumption of the neutralizing antibodies, to a switch in the epitope antibody repertoire or to an altered ratio of cytokine neutralizing to cytokine enhancing antibodies .
Air Battle Force by Dale Brown