By Jeremy Robinson
A phenomenon referred to as crustal displacement shifts the Earth's crust, repositioning continents and inflicting numerous deaths. within the wake of the worldwide disaster, the area struggles to maintain its displaced billions. yet Antarctica, freshly thawed and blooming, has emerged as a brand new desire. instead of salary an international struggle no state can suffer, the top international locations devise a contest, a race to the guts of Antarctica, with the 3 victors dividing the continent. it truly is inside this race that Mirabelle Whitney, one of many few surviving specialists at the continent, grouped with an American designated forces unit, unearths herself. however the risks waiting for the group are a long way worse than feared; past the bitter heritage of a torn kin, past the nefarious intentions in their human enemies, past the traditional creatures reborn via anhydrobiosis-there are the Nephilim. the area races to say a brand new continent, merely to discover it already taken.
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Extra info for Antarktos Rising - A Novel
On the other hand, the cytotoxic activity to K562 was slightly reduced (10--20%), as compared with control cultures (Fig. 4). These results show that endogenously produced IFN--y is essential to the proliferation and cytotoxicity of MLC but not to the lytic activity of natural killer cells, which is responsible for K562-cell killing. By using affinity-purified human antibodies to IFN--y we definitively demonstrated their unique ability to neutralize certain immunomodulating activities of IFN--y but not its antiviral and antiproliferative activity.
The characterization of human anti-cytokine autoantibodies revealed that they are polyclonal in nature and belong almost exclusively to the IgG class (14, 15). Those directed to IL-IO' are for almost 50% of the IgG4 subclass whereas anti-IFN-, and anti-IL6 autoantibodies were characterized as IgG I . Importantly, the serum levels of ACAs can vary significantly during infectious diseases with high levels at the height of the disease and a significant reduction after disease resolution (16). This suggests that when a specific cytokine exceeds a critical treshold level, ACAs may be compensatorily increased.
ALLOTRANSPLANT REJECTION Rejection of tumor, skin and heart allografts delayed by mABs anti-IFN--y. KAPOSI'S SARCOMA (KS) IFN--y induces normal endothelial cells to acquire the phenotype of KS cells. KS lesions blocked by mABs anti-IFN-T flow cytometric assay [17-18]. It remains yet unclear why the neutralizing effects of the immunoglobulins disappeared 13 days after infection, but this may be related either to preferential consumption of the neutralizing antibodies, to a switch in the epitope antibody repertoire or to an altered ratio of cytokine neutralizing to cytokine enhancing antibodies .
Antarktos Rising - A Novel by Jeremy Robinson